smithy; and of the chapel of St Serf, the patron saint of the burgh, only the tower remains. The chief industries are the manufacture of bed and table linen, towelling and woollen cloth, shipbuilding and flax-spinning. There is a steady export of coal, and the harbour is provided with a wet dock and patent slip. In smuggling days the “canty carles” of Dysart were professed “free traders.” In the 15th and 16th centuries the town was a leading seat of the salt industry (“salt to Dysart” was the equivalent of “coals to Newcastle”), but the salt-pans have been abandoned for a considerable period. Nail-making, once famous, is another extinct industry. During the time of the alliance between Scotland and Holland, which was closer in Fifeshire than in other counties, Dysart became known as Little Holland. To the west of the town is Dysart House, the residence of the earl of Rosslyn. With Burntisland and Kinghorn Dysart forms one of the Kirkcaldy district group of parliamentary burghs. The town is mentioned as early as 874 in connexion with a Danish invasion. Its name is said to be a corruption of the Latin desertum, “a desert,” which was applied to a cave on the seashore occupied by St Serf. In the cave the saint held his famous colloquy with the devil, in which Satan was worsted and contemptuously dismissed. From James V. the town received the rights of a royal burgh. In 1559 it was the headquarters of the Lords of the Congregation, and in 1607 the scene of the meetings of the synod of Fife known as the Three Synods of Dysart. Ravensheugh Castle, on the shore to the west of the town, is the Ravenscraig of Sir Walter Scott’s ballad of “Rosabelle.”
William Murray, a native of the place, was made earl of Dysart in 1643, and his eldest child and heir, a daughter, Elizabeth, obtained in 1670 a regrant of the title, which passed to the descendants of her first marriage with Sir Lionel Tollemache, Bart., of Helmingham; she married secondly the 1st duke of Lauderdale, but had no children by him, and died in 1698. This countess of Dysart (afterwards duchess of Lauderdale) was a famous beauty of the period, and notorious both for her amours and for her political influence. She was said to have been the mistress of Oliver Cromwell, and also of Lauderdale before her first husband’s death, and was a leader at the court of Charles II. Wycherley is supposed to have aimed at her in his Widow Blackacre in the Plain Dealer. Her son, Lionel Tollemache (d. 1727), transmitted the earldom to his grandson Lionel (d. 1770), whose sons Lionel (d. 1799) and Wilbraham (d. 1821) succeeded; they died without issue, and their sister Louisa (d. 1840), who married John Manners, an illegitimate son of the second son of the 2nd duke of Rutland, became countess in her own right, being succeeded by her grandson (d. 1878), and his grandson, the 8th earl.
The earldom of Dysart must not be confounded with that of Desart (Irish), created (barony 1733) in 1793, and held in the Cuffe family, who were originally of Creech St Michael, Somerset, the Irish branch dating from Queen Elizabeth’s time.
DYSENTERY (from the Gr. prefix δυσ-, in the sense of “bad,” and ἔντερον, the intestine), also called “bloody flux,” an infectious disease with a local lesion in the form of inflammation and ulceration of the lower portion of the bowels. Although at one time a common disease in Great Britain, dysentery is now very rarely met with there, and is for the most part confined to warm countries, where it is the cause of a large amount of mortality. (For the pathology see Digestive Organs.)
Recently considerable advance has been made in our knowledge of dysentery, and it appears that there are two distinct types of the disease: (1) amoebic dysentery, which is due to the presence of the amoeba histolytica (of Schaudinn) in the intestine; (2) bacillary dysentery, which has as causative agent two separate bacteria, (a) that discovered by Shiga in Japan, (b) that discovered by Flexner in the Philippine Islands. With regard to the bacillary type, at first both organisms were considered to be identical, and the name bacillus dysenteriae was given to them; but later it was shown that these bacilli are different, both in regard to their cultural characteristics and also in that one (Shiga) gives out a soluble toxin, whilst the other has so far resisted all efforts to discover it. Further, the serum of a patient affected with one of the types has a marked agglutinative power on the variety with which he is infected and not on the other.
Clinically, dysentery manifests itself with varying degrees of intensity, and it is often impossible without microscopical examination to determine between the amoebic and bacillary forms. In well-marked cases the following are the chief symptoms. The attack is commonly preceded by certain premonitory indications in the form of general illness, loss of appetite, and some amount of diarrhoea, which gradually increases in severity, and is accompanied with griping pains in the abdomen (tormina). The discharges from the bowels succeed each other with great frequency, and the painful feeling of pressure downwards (tenesmus) becomes so intense that the patient is constantly desiring to defecate. The matters passed from the bowels, which at first resemble those of ordinary diarrhoea, soon change their character, becoming scanty, mucous or slimy, and subsequently mixed with, or consisting wholly of, blood, along with shreds of exudation thrown off from the mucous membrane of the intestine. The evacuations possess a peculiarly offensive odour characteristic of the disease. Although the constitutional disturbance is at first comparatively slight, it increases with the advance of the disease, and febrile symptoms come on attended with urgent thirst and scanty and painful flow of urine. Along with this the nervous depression is very marked, and the state of prostration to which the patient is reduced can scarcely be exceeded. Should no improvement occur death may take place in from one to three weeks, either from repeated losses of blood, or from gradual exhaustion consequent on the continuance of the symptoms, in which case the discharges from the bowels become more offensive and are passed involuntarily.
When, on the other hand, the disease is checked, the signs of improvement are shown in the cessation of the pain, in the evacuations being less frequent and more natural, and in relief from the state of extreme depression. Convalescence is, however, generally slow, and recovery may be imperfect—the disease continuing in a chronic form, which may exist for a variable length of time, giving rise to much suffering, and not unfrequently leading to an ultimately fatal result.
The dysentery poison appears to exert its effects upon the glandular structures of the large intestine, particularly in its lower part. In the milder forms of the disease there is simply a congested or inflamed condition of the mucous membrane, with perhaps some inflammatory exudation on its surface, which is passed off by the discharges from the bowels. But in the more severe forms ulceration of the mucous membrane takes place. Commencing in and around the solitary glands of the large intestine in the form of exudations, these ulcers, small at first, enlarge and run into each other, till a large portion of the bowel may be implicated in the ulcerative process. Should the disease be arrested these ulcers may heal entirely, but occasionally they remain, causing more or less disorganization of the coats of the intestines, as is often found in chronic dysentery. Sometimes, though rarely, the ulcers perforate the intestines, causing rapidly fatal inflammation of the peritoneum, or they may erode a blood vessel and produce violent haemorrhage. Even where they undergo healing they may cause such a stricture of the calibre of the intestinal canal as to give rise to the symptoms of obstruction which ultimately prove fatal. One of the severest complications of the disease is abscess of the liver, usually said to be solitary, and known as tropical abscess of the liver, but probably is more frequently multiple than is usually thought.
Treatment.—Where the disease is endemic or is prevailing epidemically, it is of great importance to use all preventive measures, and for this purpose the avoidance of all causes likely to precipitate an attack is to be enjoined. Exposure to cold after heat, the use of unripe fruit, and intemperance in eating and drinking should be forbidden; and the utmost care taken as to the quality of the food and drinking water. In houses or hospitals where cases of the disease are under treatment, disinfectants should be freely employed, and the evacuations of the
