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deliver the Croonian Lecture before the Royal Society, illustrates the same thing. I received blood from the throat of an ox into two similar open earthen jars (gallipots), and slowly moved a clean glass stirring rod through the blood of one of them for a second or two, and then left both vessels undisturbed.
In the course of a few minutes the blood that had been thus gently and briefly stirred was a mass of coagulum, while the unstirred blood was still fluid, except a thin layer of clot encrusting the wall of the jar. In course of time it also coagulated completely. Now we know, from the experiment with the ox's jugular, that coagulation is propagated with extreme slowness, if at all, from a clot in blood perfectly undisturbed. The earlier coagulation of the main mass of the stirred blood was, therefore, not caused by propagation of the process from a layer upon the surface of the jar, but must have been the result of the brief agency of the glass rod.
A little before the delivery of the lecture referred to,[1] I became aware of the recent very important observations of Schmidt, who showed, as had been foreshadowed many years previously by Andrew Buchanan, of Glasgow, that normal liquor sanguinis does not, as had been supposed, contain fibrin in solution, but only one constituent of that substance, termed by Schwann Fibrinogen,
- ↑ Vide the Croonian Lecture on the "Coagulation of the Blood," "Proceedings of the Royal Society," 1863.
