Page:Cellular pathology as based upon physiological and pathological histology.djvu/30

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CONTENTS.
LECTURE XII.—The Nervous System. 280
Peripheral terminations of the nerves. Nerves of special sense. The skin and the distinction of vessel-, nerve-, and cell-territories in it. Olfactory mucous membrane. Retina. Division of nerve-fibres. The electrical organ of fishes. Muscles. Further consideration of nerve-territories.—Nervous plexuses with ganglioniform enlargements. Intestines.—Errors of the neuro-pathologists.—The great nervous centres. Grey substance. Ganglion- [nerve-] cells containing pigment. Varieties of ganglion-cells; sympathetic cells in the spinal marrow and brain, motor and sensitive cells. Multipolar (polyclonous) ganglion-cells. Different nature of the processes of ganglion-cells.  
LECTURE XIII.—Spinal Cord and Brain. 302
The spinal cord. White and grey matter. Central canal. Groups of ganglion-cells. White columus and commissures.—The medulla oblongata and the brain. Its granular and bacillar layer.—The spinal cord of the petromyzon and its non-medullated fibres.—The intermediate substance (interstitial tissue). Ependyma ventriculorum. Neuro-glia. Corpora amylacea.  
LECTURE XIV.—Activity and Irritability of Cellular Elements. Different Forms of Irritation. 321
Life of individual parts. The unity of the neurists. Consciousness. Activity of individual parts. Excitability (irritability) as a general criterion of life. Meaning of irritation. Partial death. Necrosis.—Function, nutrition, and formation, as general forms of vital activity. Difference of irritability according to the different forms of activity.—Functional irritability. Nerves, muscles, ciliated epithelium, glands. Fatigue and functional restitution. Stimuli. Their specific relations. Muscular irritability.—Nutritive irritability. Maintenance and destruction of elements. Inflammation. Cloudy swelling. Kidney (morbus Brightii) and cartilage. Neuro-pathological doctrines. Skin, cornea. The humoro-pathological doctrines. Parenchymatous exudation, and parenchymatous inflammation.—Formative irritation. Multiplication of nucleoli and nuclei by division. Multi-nuclear cells; marrow-cells and myeloid tumours. Comparison between formative muscular irritation and muscular growth. Multiplication (new formation) of cells by division. The humoro- and neuro-pathological doctrines.—Inflammatory irritation as a compound phenomenon. Neuro-paralytical inflammation (Vagus, Trigeminus).  
LECTURE XV.—Passive Processes. Fatty Degeneration. 356
Passive processes in their two chief tendencies to degeneration; Necrobiosis (softening and disintegration) and induration.—Fatty degeneration. Histological history of fat in the animal body; fat as a component of the tissues, as a transitory infiltration, and as a necrobiotic matter.—Adipose tissue. Polysarcia. Fatty tumours. Interstitial formation of fat. Fatty